I don't think I said smoking weed causes other people to try hard drugs If I did I didn't mean to. What I did mean was that people who smoke weed (especially at earlier ages) are more likely to experiment with hard drugs and maybe become drug addicts. There are plenty of studies and statistics to support it. Like below.
My opinion is that people who go on try hard drugs just try marijuana first because it's cheap and easily attained. If it wasn't, they would probably try hard drugs anyway. |
That could be true for some, but when people get into smoking weed because it is cheap and easy to get they also start opening up doors and connections to obtain more harder drugs. They start to get into the drug scene.
That's different, but other studies disagree: |
Could you link me a study that says use of weed does not increase the risk of use of harder drugs? Here are some that do..
Results Individuals who used cannabis by age 17 years had odds of other drug use, alcohol dependence, and drug abuse/dependence that were 2.1 to 5.2 times higher than those of their co-twin, who did not use cannabis before age 17 years. Controlling for known risk factors (early-onset alcohol or tobacco use, parental conflict/separation, childhood sexual abuse, conduct disorder, major depression, and social anxiety) had only negligible effects on these results. These associations did not differ significantly between monozygotic and dizygotic twins.
Conclusions Associations between early cannabis use and later drug use and abuse/dependence cannot solely be explained by common predisposing genetic or shared environmental factors. The association may arise from the effects of the peer and social context within which cannabis is used and obtained. In particular, early access to and use of cannabis may reduce perceived barriers against the use of other illegal drugs and provide access to these drugs.
http://jama.jamanetwork.com/article.aspx?articleid=195839
http://www.abc.net.au/news/2007-07-18/cannabis-linked-to-use-of-amphetamines/2505708
And here is some more studies
The gateway model was developed to explain a number of statistical sets that showed a correlation between the use of marijuana, alcohol, and tobacco, and the use of so-called “harder” drugs (heroin, cocaine, LSD, etc.) in adolescents
(Morral et al., 2002). The marijuana gateway effect refers specifically to marijuana’s contribution to this general gateway effect. These statistics generally fit into three different subsets, all of which are used to support the marijuana gateway effect. The first simply demonstrate that users of marijuana will be at a higher risk to experiment with hard drugs than non-users. This is supported by results from a number of studies in a number of different countries and cultures
(Adler & Kandel, 1981; Stenbacka, Allebeck & Romelsjo, 1993; Beenstock & Rahav, 2002). The second statistical set is related to the order in which different classes of drugs are first used. Drug abuse rarely begins with hard drugs and regresses to marijuana. A longitudinal study of 1265 New Zealand adolescents between 15 and 21 years of age revealed only three cases (.237 percent) in which hard drug use was initiated before marijuana use
(Fergusson & Horwood, 2000). The third factor used to support the marijuana gateway effect is the strong correlation between the frequency of marijuana use and the rate of hard drug initiation
(Morral et al., 2002). A proportional hazards model implied that those using marijuana more than 50 times per year were 140 times more likely to progress to hard drugs than those test subjects using no cannabis
(Fergusson & Horwood, 2000). This correlation between number of marijuana usage and progression to harder drugs has been labeled the dose-response effect
(Morral et al., 2002). These three relationships between marijuana and hard drug use would suggest that the marijuana gateway effect has a solid foundation in science.